EGCG inhibits cardiomyocyte apoptosis in pressure overload‐induced cardiac hypertrophy and protects cardiomyocytes from oxidative stress in rats

Acute EGCG Supplementation Reverses Endothelial Dysfunction in Patients with Coronary Artery Disease

EGCG, a green tea polyphenol, improves endothelial function and insulin sensitivity, reduces blood pressure, and protects against myocardial I/R injury in SHR

Molecular understanding of Epigallocatechin gallate (EGCG) in cardiovascular and metabolic diseases

In Vitro Antioxidant effect of Green Tea Polyphenol Epigallocatechin-3-Gallate (EGCG) in Protecting Cardiovascular Diseases

EGCG protects against oxidized LDL-induced endothelial dysfunction by inhibiting LOX-1-mediated signaling

Antihyperglycemic, Antidyslipidemic and Antifibrotic effect of EGCG in STZ-High Fat Diet Induced DCM rats

Molecular identification for epigallocatechin-3-gallate-mediated antioxidant intervention on the H2O2-induced oxidative stress in H9c2 rat cardiomyoblasts

Epigallocatechin-3-gallate-mediated cardioprotection by Akt/GSK-3β/caveolin signalling in H9c2 rat cardiomyoblasts

Attenuation of the cardiac inflammatory changes and lipid anomalies by (−)-epigallocatechin-gallate in cigarette smoke-exposed rats

Absorption, metabolism and antioxidative effects of tea catechin in humans

Protective Effects of Green Tea Catechin on Cardio- and Cerebral Vascular Diseases

Epigallocatechin-3-O-Gallate, a Green Tea Polyphenol, Induces Expression of Pim-1 Kinase Via PPARγ in Human Vascular Endothelial Cells

EGCG, a green tea polyphenol, improves endothelial function and insulin sensitivity, reduces blood pressure and protects against myocardial I/R injury in SHR

Targeting cell signaling and apoptotic pathways by EGCG: A potential role in the prevention and treatment of ischemic heart disease

Epigallocatechin-3-gallate protects against diabetic cardiomyopathy through modulating the cardiometabolic risk factors, oxidative stress, inflammation, cell death and fibrosis in streptozotocin-nicotinamide-induced diabetic rats

Epigallocatechin-3 Gallate, a Green Tea Catechin, Attenuated the Downregulation of the Cardiac Gap Junction Induced by High Glucose in Neonatal Rat Cardiomyocytes

Mitogenesis of Vascular Smooth Muscle Cell Stimulated by Platelet-Derived Growth Factor-bb Is Inhibited by Blocking of Intracellular Signaling by Epigallocatechin-3-O-Gallate

Epigallocatechin-3-gallate, the major green tea catechin, regulates the desensitization of β1 adrenoceptor via GRK2 in experimental heart failure

Epigallocatechin-3-gallate induces contraction of the rat aorta by a calcium influx-dependent mechanism

Acute vascular and metabolic actions of the green tea polyphenol epigallocatechin 3-gallate in rat skeletal muscle

Epigallocatechin Gallate Reduces Ischemia/Reperfusion Injury in Isolated Perfused Rabbit Hearts

Epigallocatechin Gallate Promotes MicroRNA 145 Expression against Myocardial Hypoxic Injury through Dab2/Wnt3a/β-catenin

Green and black tea are equally potent stimuli of NO production and vasodilation: new insights into tea ingredients involved

Oral administration of green tea Epigallocatechin-3-gallate reduces oxidative stress and enhances restoration of cardiac function in diabetic rats receiving autologous transplantation of adipose-derived stem cells